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Hard Drugs!!! Serious replies only


Swatkat

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E db lo evarina drugs teskunaara?

reasons enti

nakithey nidrapoi lesthey vache feeling enni milliona ichina raadhu i enjoy afternoons naps ithey inka super after lunch! 
 

endhuku teskuntaru drugs?

1) saradaga friends tho elli habit forming ah?

2) teskunn atarvtha ah feeling ela untadhi deep sleep tarvtha vache calming affect kantey ekuva pleasure isthadha?

3) nidra pattaka teskuntaara? I dont think so adhi stimulant

4) excitement for no reason vasthada?

5) ah mathulo opportunities dorkuthaye ani velthara?

6) lekapothey jeevitham lo anni chusesi inka edho exp avalanai teskuntara?

 

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  • Swatkat changed the title to Hard Drugs!!! Serious replies only
16 minutes ago, Swatkat said:

@siva604 only doctor vi nuvve ekina experience undha patients tho matladetapudu i know ilanti psy ward lo ekuva untaye

Drug dependency is really bad thing..Most of the people hooked to it for fun and it is tough for them to come off of it. Celebrities [ Performance anxiety..lets stay rock stars who are under sheer pressure etc.. thats why you see most of them dying young. once you have 50 million $ with you when you are in mid 20s money is not purpose anymore.. they want to produce content, competition and live a second life etc]

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The primary factor in the development of addiction is neurophysiologic reinforcement (reward). One specific mesolimbic "reward pathway" has been identified in the brain31, and others may exist. This pathway involves dopaminergic neurons that originate in the ventral tegmental area (VTA) and project into the forebrain, particularly the nucleus accumbens (See Figure 2 below). The dopaminergic neurons are probably under continuous inhibition in the VTA, perhaps by gamma-aminobutyric acid (GABA). Release of dopamine from these neurons onto the dopamine receptors in the nucleus accumbens produces positive reinforcement.

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diagnosisbrains.gif

 

Animal studies suggest the existence of at least one central reward-reinforcement pathway for drug self-administration in the human brain. The stimulant-reward system (top) directly influences the neurons using dopamine that connect from the ventral tegmental area (VTA) to the nucleus accumbens (NA) and thereafter to the frontal cortex. The opioid system (middle) appears to involve structures such as the periaqueductal gray area, arcuate nucleus, amygdala and locus coeruleus, which use peptides that mimic the action of drugs such as heroin and morphine and indirectly influence the NA in a manner similar to that of stimulants. Alcohol and other sedative-hypnotics (bottom) also indirectly affect the VTA-NA reward system. This effect is mediated by GABA receptors, which are distributed widely and which influence the central dopaminergic reward system through mechanisms that include opioid pathways. (Adapted from Brain concepts: drugs and the brain. Washington, DC: Society for Neuroscience, 1992.)

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